Early enhanced Th1 response after Leishmania amazonensis infection of C57BL/6 interleukin-10-deficient mice does not lead to resolution of infection
Abstract
C3H and C57BL/6 mice are resistant to Leishmania major but develop chronic lesions with persistent parasite loads when they are infected with Leishmania amazonensis. These lesions develop in the absence of interleukin-4 (IL-4), indicating that susceptibility to this parasite is not a result of devel …
Keywords
National Center for Biotechnology Information
NCBI
NLM
MEDLINE
Mice
Inbred C57BL
animals
pubmed abstract
nih
national institutes of health
national library of medicine
research support
non-u.s. gov't
u.s. gov't
P.H.S.
Antibodies
hypersensitivity
immunoglobulin g / classification
interferon-gamma / biosynthesis
delayed
protozoan / biosynthesis
leishmaniasis
cutaneous / immunology*
th1 cells / immunology*
nitric oxide synthase / genetics
nitric oxide synthase type ii
interleukin-10 / physiology*
interleukin-12 / biosynthesis
pmid:11895981
pmc127855
doi:10.1128/iai.70.4.2151-2158.2002
douglas e jones
mark r ackermann
phillip scott
leishmania major
leishmania mexicana / immunology*
cutaneous / pathology
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Citation
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274226
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