Inactivation of TGFβ signaling in neural crest stem cells leads to multiple defects reminiscent of DiGeorge syndrome
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Inactivation of TGFβ signaling in neural crest stem cells leads to multiple defects reminiscent of DiGeorge syndrome

Heiko Wurdak, Lars M. Ittner, Karl S. Lang, Per Leveen, Ueli Suter, Jan A. Fischer, Stefan Karlsson, Walter Born, Lukas Sommer;Heiko Wurdak;Lars M. Ittner;Karl S. Lang;Per Leveen;Ueli Suter;Jan A. Fischer;Stefan Karlsson;Walter Born;Lukas Sommer;
genes & development 2005 Vol. 19 pp. 530-
18
sommer2005genesinactivation

Abstract

Specific inactivation of TGFβ signaling in neural crest stem cells (NCSCs) results in cardiovascular defects and thymic, parathyroid, and craniofacial anomalies. All these malformations characterize DiGeorge syndrome, the most common microdeletion ...

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DOI:
10.1101/gad.317405
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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC551573...

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