Resveratrol protects cardiomyocytes against anoxia/reoxygenation via dephosphorylation of VDAC1 by Akt-GSK3 β pathway
Abstract
Our previous studies showed that the effect of resveratrol preventing mitochondrial permeability transition pore (mPTP) opening in myocardial ischemia/reperfusion injury was achieved by regulating voltage-dependent anion channel 1 (VDAC1). However, the underlying mechanism remains unclear. Previous …
Keywords
National Center for Biotechnology Information
NCBI
NLM
MEDLINE
animals
pubmed abstract
nih
national institutes of health
national library of medicine
newborn
pmid:30445019
doi:10.1016/j.ejphar.2018.11.016
Mengyuan Tian
Yongyan Xie
Zhangping Liao
Apoptosis / drug effects
Cardiotonic Agents / pharmacology*
Cell Hypoxia / drug effects
Cells
Cultured
Glycogen Synthase Kinase 3 beta / metabolism
Myocytes
Cardiac / drug effects*
Cardiac / physiology
Phosphorylation / drug effects
Proto-Oncogene Proteins c-akt / metabolism
Rats
Sprague-Dawley
Resveratrol / pharmacology*
Voltage-Dependent Anion Channel 1 / metabolism
Citation
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110499
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