PTEN mutations do not cause nuclear beta-catenin accumulation in endometrial carcinomas
Abstract
PTEN: and beta-catenin mutations constitute the predominant genetic alterations in endometrioid carcinomas of the endometrium. PTEN encodes a dual-specificity phosphatase with lipid phosphatase and protein tyrosine phosphatase activities that regulate both apoptosis and interactions with the extrace …
Keywords
National Center for Biotechnology Information
NCBI
NLM
MEDLINE
humans
pubmed abstract
nih
national institutes of health
national library of medicine
research support
non-u.s. gov't
female
carcinoma
immunohistochemistry
mutation*
trans-activators / metabolism*
cell nucleus / metabolism
pten phosphohydrolase
beta catenin
pmid:15492994
doi:10.1016/j.humpath.2004.06.007
b wappenschmidt
e wardelmann
r k schmutzler
endometrioid / genetics*
endometrioid / metabolism*
endometrioid / pathology
cytoskeletal proteins / metabolism*
dna mutational analysis
endometrial neoplasms / genetics*
endometrial neoplasms / metabolism*
endometrial neoplasms / pathology
phosphoric monoester hydrolases / genetics*
tumor suppressor proteins / genetics*
Citation
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266810
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